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Videonystagmography in one page

For clinic. Not a substitute for the full atlas.
Normal values
Spontaneous nystagmus SPV< 6°/s
VOR gain (vHIT)≥ 0.8
Unilateral weakness< 25%
Directional preponderance< 30%
Fixation index (peripheral)< 0.5
Saccade peak velocity (20°)> 300°/s
Saccade latency~200 ms
Smooth pursuit gain (< 0.4 Hz)> 0.8
Jongkees formulas
Unilateral weakness (UW)
|(RW + RC) − (LW + LC)|
÷ (RW + RC + LW + LC) × 100
Threshold ≥ 25%. Tells you which ear is weak.
Directional preponderance (DP)
|(RW + LC) − (RC + LW)|
÷ (RW + RC + LW + LC) × 100
Threshold ~30%. Asymmetry of direction, less specific.
Caloric mnemonics
COWS
Cold Opposite, Warm Same
(refers to fast-phase direction)

Body position
Supine, head elevated 30°
(horizontal canal vertical)

Frequencies tested
Caloric ≈ 0.003 Hz
Rotational chair ≈ 0.01–0.6 Hz
vHIT ≈ 5 Hz
HINTS exam — acute vestibular syndrome
ComponentPeripheral patternCentral pattern (HINTS+)
Head ImpulsePositive (corrective saccade on affected side)Normal (no saccade)
NystagmusDirection-fixed (Alexander's law)Direction-changing with gaze
Test of SkewAbsentSkew present
One central feature is enough to require imaging. HINTS+ is more sensitive than early MRI for posterior circulation stroke. Requires acute, persistent vertigo and competent examination technique.
Slow-phase shape → localisation
Slow phase shapeSuggestsClassic associations
Decreasing-velocity (decelerating)Acute peripheralVestibular neuritis, labyrinthitis
LinearCompensated peripheral or centralCheck fixation suppression
Increasing-velocity (accelerating)Ocular-motor / congenitalInfantile nystagmus syndrome (INS)
Envelope (crescendo-decrescendo)BPPV canalithiasisPosterior canal > horizontal canal
Sustained, no latencyBPPV cupulolithiasisTreat with Semont liberatory
Pendular (sinusoidal)Congenital or centralMS, brainstem stroke, oculopalatal myoclonus
Periodic reversal (~90–120 s)Central — vestibulocerebellumPAN. Often baclofen-responsive
BPPV at a glance
FeatureCanalithiasisCupulolithiasis
Latency1–5 sNone
Duration< 60 s, crescendo-decrescendoSustained while in position
FatigueYes, with repetitionNo
TreatmentEpley (posterior) · BBQ roll (horizontal)Semont liberatory
Posterior canal ≈ 85–90% of BPPV. Horizontal canal ≈ 10%. Anterior canal < 2%.
Episodic vertigo — timing & clues
DurationBest fitKey clue
< 1 minBPPVTriggered by position change
MinutesVestibular paroxysmia · TIASudden, brief, recurrent
20 min – 24 hMénière diseaseLow-freq SNHL, fullness, tinnitus
5 min – 72 hVestibular migrainePhotophobia/phonophobia, migraine hx
Days–weeksVestibular neuritis · strokeAcute, persistent → HINTS
Red flags — image first
  • Pure vertical or pure torsional nystagmus in primary gaze
  • Nystagmus that does not suppress with fixation (FI ≥ 0.7)
  • Direction-changing nystagmus with gaze
  • Skew deviation or vertical misalignment
  • Normal head-impulse + acute persistent vertigo
  • Down-beat nystagmus in primary gaze
  • Up-beat nystagmus + altered mental status (suspect Wernicke)
  • Acute vertigo + new neurological deficit
  • Sudden onset in patient with vascular risk factors
  • Pendular nystagmus in an adult (acquired)
Distilled from McCaslin (2013), Leigh & Zee (2006), Jongkees (1962), Bhattacharyya et al. (2017).
For educational use only. Not a clinical decision aid. Clinicians retain full responsibility for interpretation, diagnosis, and management.
Concept & Design
Dr. Prahlada N. B
Champions Educational and Medical Society (R)
& Amogh Foundation